Background: Physical exercise has many benefits for the body. Physical Exercise according to the procedure has a good effect on the body. However, high-intensity exercise that is carried out continuously will produce quite a lot of free radicals in the body and cause the body to experience oxidative stress. Oxidative stress that is not handled properly can cause cell damage and accelerate aging. To determine the presence of oxidative stress in the body, it can be measured by checking the levels of superoxide dismutase (SOD) and also malondialdehyde (MDA). One way to prevent oxidative stress in the body is to consume antioxidant-rich foods such as purple sweet potatoes. Purple Sweet Potato contains anthocyanins and ascorbic acid which can neutralize free radicals and prevent oxidative stress. Objective: This study was conducted to determine the effect of purple sweet potato consumption on oxidative stress after high-intensity exercise. Methods: Experimental research with post-test only control group design. The number of samples was 24 rats divided into 4 groups, 1 group consisting of 6 rats. Group I was only given standard feed, group II was given swimming for ±25 minutes and standard feed. Meanwhile, group III was given swimming training, standard feed and purple sweet potato 1.6 mg/day. Group IV was given swimming training, standard feed and purple sweet potato 3.6 mg/day. Physical exercise and dietary purple sweet potato was given every day for 28 days. Purple Sweet Potatoes are processed by steaming and then put into a juicer machine. Observation of SOD and MDA levels by spectrophotometry on day 29. Results: Analysis using one-way ANOVA and post hoc LSD showed that purple sweet potato at a dose of 3.6 mg/day was the most effective dose and had a significant effect on decreasing MDA levels (p<0 0.05), and also had a significant effect on increasing SOD activity (p<0.05). Conclusion: Giving purple sweet potato at a dose of 3.6 mg/day in rats or the equivalent of 200gram/day in humans can prevent the accumulation of free radicals that cause oxidative stress in the body.